regM appears to be a histidine protein kinase that is reiterated in the genome, thus a double mutant must be constructed before further analysis. regR appears to be a hybrid histidine kinase-response regulator. The phenotype of regR mutants displayed no discernible difference from wild-type phenotype. However, RegR appears to play a role in transcription regulation. Of the 1380 mutants screened in the gusA screen, 37 mutants appear to be positively regulated and 87 mutants appear to be negatively regulated.
Was this article helpful?