Results and Discussion

regM appears to be a histidine protein kinase that is reiterated in the genome, thus a double mutant must be constructed before further analysis. regR appears to be a hybrid histidine kinase-response regulator. The phenotype of regR mutants displayed no discernible difference from wild-type phenotype. However, RegR appears to play a role in transcription regulation. Of the 1380 mutants screened in the gusA screen, 37 mutants appear to be positively regulated and 87 mutants appear to be negatively regulated.

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